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By Vinood B. Patel, Victor R. Preedy (eds.)

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Extra resources for Biomarkers in Kidney Disease

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Biomarkers of Allograft Rejection . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . Biomarkers of Tolerance . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . Gene Expression Studies . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . Potential Applications to Prognosis, Other Diseases, or Conditions . . .

SCD30 levels among patients with BPAR were compared with those of patients with stable graft function and non-rejection cause of acute allograft dysfunction (including CMV infection, ATN, and calcineurin inhibitor toxicity). 7 %, respectively. It could not predict the 2-year graft survival. Pretransplant sCD30 level could not predict acute rejection, and there was a significant elevation in sCD30 level during the episodes of BPAR. Thus, sCD30 level after transplantation and its changes could be used as a predictor of acute rejection (Nafar et al.

5 5 5 5 6 7 19 19 22 22 24 Abstract Kidney transplantation is the optimal renal replacement therapy. The progressions in immunosuppressive drugs improved the short-term survival, but 10-year graft survival is about 50 %, only. Acute or chronic rejection, drug nephrotoxicity, and transplant glomerulopathy all have adverse impacts on graft survival. Most of these events are the result of over- or under-immunosuppression.

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